Acute Gastritis (AG)
Acute erosive gastritis typically involves discrete foci of surface necrosis due to damage to mucosal defenses. NSAIDs inhibit cyclooxygenase-1, or COX-1, an enzyme responsible for the biosynthesis of eicosanoids in the stomach, which increases the possibility of peptic ulcers forming. Also, NSAIDs, such as aspirin, reduce a substance that protects the stomach called prostaglandin. These drugs used in a short period are not typically dangerous. However, regular use can lead to gastritis. Additionally, severe physiologic stress from sepsis, hypoxia, trauma, or surgery, is also a common etiology for acute erosive gastritis. This form of gastritis can occur in more than 5% of hospitalized patients.
Organism species: Pan-species (General)
PRODUCT TYPE | CATALOG NO. | PRODUCT NAME | APPLICATIONS |
---|---|---|---|
Proteins | N/A | Recombinant Acute Gastritis (AG) | Antigenic Transformation Customized Service Offer |
Antibodies | N/A | Monoclonal Antibody to Acute Gastritis (AG) | Monoclonal Antibody Customized Service Offer |
N/A | Polyclonal Antibody to Acute Gastritis (AG) | Polyclonal Antibody Customized Service Offer | |
Assay Kits | N/A | CLIA Kit for Acute Gastritis (AG) | CLIA Kit Customized Service Offer |
N/A | ELISA Kit for Acute Gastritis (AG) | ELISA Kit Customized Service Offer |